12/13/2023 0 Comments Plexiform layers![]() ĭemirkaya N, Wit FW, van Den Berg TJ, Kooij KW, Prins M, Schlingemann RO, Abramoff MD, Reiss P, Verbraak FD (2016) HIV-associated neuroretinal disorder in patients with well-suppressed HIV-infection: a comparative cohort study. Spaide RF, Klancnik JM Jr, Cooney MJ (2015) Retinal vascular layers imaged by fluorescein angiography and optical coherence tomography angiography. Lagathu C, Cossarizza A, Bereziat V, Nasi M, Capeau J, Pinti M (2017) Basic science and pathogenesis of ageing with HIV: potential mechanisms and biomarkers. Kozak I, Sasik R, Freeman WR, Sprague LJ, Gomez ML, Cheng L, El-Emam S, Mojana F, Bartsch DU, Bosten J, Ayyagari R, Hardiman G (2013) A degenerative retinal process in HIV-associated non-infectious retinopathy. Shah KH, Holland GN, Yu F, Van Natta M, Nusinowitz S (2006) Contrast sensitivity and color vision in HIV-infected individuals without infectious retinopathy. Kozak I, Sample PA, Hao J, Freeman WR, Weinreb RN, Lee TW, Goldbaum MH (2007) Machine learning classifiers detect subtle field defects in eyes of HIV individuals. įalkenstein I, Kozak I, Kayikcioglu O, Cheng L, Bartsch DU, Azen SP, Labree LD, Freeman WR (2006) Assessment of retinal function in patients with HIV without infectious retinitis by multifocal electroretinogram and automated perimetry. Stewart MW (2017) Ophthalmologic disease in HIV infection: recent changes in pathophysiology and treatment. Sugar EA, Jabs DA, Ahuja A, Thorne JE, Danis RP, Meinert CL (2012) Incidence of cytomegalovirus retinitis in the era of highly active antiretroviral therapy. Robinson MR, Ross ML, Whitcup SM (1999) Ocular manifestations of HIV infection. Teeraananchai S, Kerr SJ, Amin J, Ruxrungtham K, Law MG (2017) Life expectancy of HIV-positive people after starting combination antiretroviral therapy: a meta-analysis. OCTA is an important test for the screening of retinal microvascular changes over time in HIV-infected cases. The results of this study provide evidence of microvascular and neuroretinal loss in individuals with well-suppressed HIV infection, compared with healthy control subjects. Using Pearson’s correlation analysis, no significant correlation was determined between the duration of HIV infection and mean GC-IPL, MT and VD, and PD values ( r − 0.223, p 0.141 r − 0.223, p 0.141 r − 0.169, p 0.268 r − 0.105, p 0.491 r − 0.095, p 0.535 respectively). There was a significant difference between the average and superior and inferior half-region of GC-IPL values. Peripapillary PD was significantly decreased in the HIV group. A significant difference was found between the two groups in respect of the mean VD and PD parameters ( p < 0.05). ![]() No statistically significant difference was determined between the groups in respect of the average and foveal MT ( p = 0.05). The mean CD4 count (nadir) for all the patients was 147.09 ± 122 cells/mm 3 and the mean RNA was 173.6 ± 913.8 copies/ml. The mean disease duration was 7.3 ± 1.9 years (range, 5–12 years) in the HIV group. ![]() Optical coherence tomography angiography (OCTA) was used for the assessment of macular, peripapillary retinal nerve fiber layer (RNFL) thicknesses, ganglion cell–inner plexiform layer, vessel density, perfusion density, and foveal avascular zone. This prospective, cross-sectional case-control study included 45 HIV-infected patients and 45 healthy individuals. To investigate the long-term effect of HIV infection on the ganglion cell –inner plexiform layer and retinal capillary network. ![]()
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